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anti-ciap1 antibodies (mouse and human)  (Cell Signaling Technology Inc)


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    Structured Review

    Cell Signaling Technology Inc anti-ciap1 antibodies (mouse and human)
    Anti Ciap1 Antibodies (Mouse And Human), supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/anti-ciap1 antibodies (mouse and human)/product/Cell Signaling Technology Inc
    Average 90 stars, based on 1 article reviews
    anti-ciap1 antibodies (mouse and human) - by Bioz Stars, 2026-03
    90/100 stars

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    Schematic presentation indicating the suggested mechanisms by which inhibit NF-κB-induced SKM-1 cell apoptosis by ATO combined with TL. The combined effect of TL and ATO inhibits the NF-κB signaling pathway, promotes the expression of p65, further inhibits the expression of IkBα, p52, and RelB, further upregulates the expression of Caspase-3, Caspase-8, PARP, downregulates the expression of Bcl-2, Bcl-XL, and <t>cIAP1,</t> and ultimately promotes SKM-1 cell apoptosis.
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    Proteintech proteintech anti ciap1
    Schematic presentation indicating the suggested mechanisms by which inhibit NF-κB-induced SKM-1 cell apoptosis by ATO combined with TL. The combined effect of TL and ATO inhibits the NF-κB signaling pathway, promotes the expression of p65, further inhibits the expression of IkBα, p52, and RelB, further upregulates the expression of Caspase-3, Caspase-8, PARP, downregulates the expression of Bcl-2, Bcl-XL, and <t>cIAP1,</t> and ultimately promotes SKM-1 cell apoptosis.
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    Cell Signaling Technology Inc anti-ciap1 antibodies (mouse and human)
    Schematic presentation indicating the suggested mechanisms by which inhibit NF-κB-induced SKM-1 cell apoptosis by ATO combined with TL. The combined effect of TL and ATO inhibits the NF-κB signaling pathway, promotes the expression of p65, further inhibits the expression of IkBα, p52, and RelB, further upregulates the expression of Caspase-3, Caspase-8, PARP, downregulates the expression of Bcl-2, Bcl-XL, and <t>cIAP1,</t> and ultimately promotes SKM-1 cell apoptosis.
    Anti Ciap1 Antibodies (Mouse And Human), supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/anti-ciap1 antibodies (mouse and human)/product/Cell Signaling Technology Inc
    Average 90 stars, based on 1 article reviews
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    Schematic presentation indicating the suggested mechanisms by which inhibit NF-κB-induced SKM-1 cell apoptosis by ATO combined with TL. The combined effect of TL and ATO inhibits the NF-κB signaling pathway, promotes the expression of p65, further inhibits the expression of IkBα, p52, and RelB, further upregulates the expression of Caspase-3, Caspase-8, PARP, downregulates the expression of Bcl-2, Bcl-XL, and cIAP1, and ultimately promotes SKM-1 cell apoptosis.

    Journal: Medicine

    Article Title: Triptolide combined with arsenic trioxide induces apoptosis of myelodysplastic syndrome cells by inhibiting the NF-κB signaling pathway

    doi: 10.1097/MD.0000000000046206

    Figure Lengend Snippet: Schematic presentation indicating the suggested mechanisms by which inhibit NF-κB-induced SKM-1 cell apoptosis by ATO combined with TL. The combined effect of TL and ATO inhibits the NF-κB signaling pathway, promotes the expression of p65, further inhibits the expression of IkBα, p52, and RelB, further upregulates the expression of Caspase-3, Caspase-8, PARP, downregulates the expression of Bcl-2, Bcl-XL, and cIAP1, and ultimately promotes SKM-1 cell apoptosis.

    Article Snippet: Western blot, qPCR, and BCA protein assay kits were obtained from Nanjing Vazyme Biotech Co., Ltd. Antibodies targeting apoptosis proteins like Bcl-2, Bcl-XL, cIAP1, Caspase-3, Caspase-8, and poly ADP-ribose polymerase (PARP), as well as those for NF-κB pathway proteins including IkBα, p65, p52, and RelB, were obtained from Proteintech Group, Inc.

    Techniques: Expressing